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Fig. 8 | BMC Plant Biology

Fig. 8

From: Integrating transcriptomic and metabolomic analysis of hormone pathways in Acer rubrum during developmental leaf senescence

Fig. 8

Biosynthetic and signal transduction pathways of brassinosteroids. 90B/724B, steroid 22-alpha-hydroxylase; 6-DeoxoCT, 6-Deoxocathasterone; 6-OHCN, 6alpha-Hydroxycampestanol; 6-DeoxoTE, 6-Deoxoteasterone; 6-OxoCN, 6-Oxocampestanol; 6-Deoxo3DT, 3-Dehydro-6-deoxoteasterone; CT, cathasterone; 6-DeoxoTY, 6-Deoxotyphasterol; TE, teasterone; 6-DeoxoCS, 6-Deoxocastasterone; 3-DT, 3-Dehydroteasterone; 6-OHCS, 6alpha-Hydroxy-castasterone; TY, Typhasterol; 85A1/2, brassinosteroid-6-oxidase 1; 92A6, typhasterol/6-deoxotyphasterol 2alpha-hydroxylase; 85A2, brassinosteroid-6-oxidase 2; BRI1, protein brassinosteroid insensitive 1; BAK1, brassinosteroid insensitive 1-associated receptor kinase 1; BSK, BR-signaling kinase; BSU1, serine/threonine-protein phosphatase; BIN2, brassinosteroid insensitive 2; BZR1/2, brassinosteroid resistant 1/2; PP2A, protein phosphatase type 2A. GL, non-senescing leaves, fully expanded green leaves; YL, senescing leaves, completely yellow; ~‚ÄČ95% chlorophyll loss. Colored cells represent the expression profiles of differentially expressed genes (DEGs). Heatmaps were generated with the Rpackage and the color bar at the lower right. Orange dots show the fold change of the content of compounds in non-senescing and senescing leaves

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