Multi-environment QTL studies suggest a role for cysteine-rich protein kinase genes in quantitative resistance to blackleg disease in Brassica napus

Table 1 Survival, internal infection and heritability of DH populations in two environments

Population	Location	Year	Blocks	Metric	Range (%)	Mean (%)	Med.(%)	σ² _A	σ² _E	h ²
Topas/	Horsham	2009	3	S	0–100	25.9	20	1199.6	148.7	0.89
AG-Castle				II	12–100	58.6	56	1616.2	277.4	0.85
	Wagga Wagga	2009	3	S	0–100	43.2	43	590.5	197	0.75
				II	12–100	56.2	56	729.5	193	0.79
	Wagga Wagga	2010	4	S	0–100	38.6	38	1106.4	165.9	0.87
				II	4–100	38.5	36	741.8	120.7	0.86
Topas/	Horsham	2008	3	S	0–71.1	11.2	6.8	419	44.5	0.90
AV-Sapphire				II	8–100	62.9	64	1937	263	0.88
	Wagga Wagga	2009	3	S	0–100	32	28	1069	223.4	0.83
				II	4–100	45.4	40	941.4	348.7	0.73
	Wagga Wagga	2011	4	S	0–100	69.2	72.8	1325.2	205.1	0.87
				II	16–100	45	44	632.2	123.4	0.84
	Horsham	2012	4	S	0–100	16.5	9	1051.8	155.7	0.87
				II	24.2–100	87.4	94	700.4	85.1	0.89

Data given for each population (Topas/AG-Castle or Topas/AV-Sapphire) in each environment (location x year)
Blocks represent replicates per trial, scoring metrics; S survival, II internal infection
Range, mean and median (Med.) given for total entries (blocks x DH lines). σ² _A = variance (additive), σ² _E = variance (environmental); h ² = heritability (σ² _A/σ² _A + σ² _E)

ISSN: 1471-2229