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Fig. 7 | BMC Plant Biology

Fig. 7

From: Linking genome wide RNA sequencing with physio-biochemical and cytological responses to catalogue key genes and metabolic pathways for alkalinity stress tolerance in lentil (Lens culinaris Medikus)

Fig. 7

Schematic representation of (a) overall putative differentially expressed genes (DEGs) and modulated pathways in response to alkalinity stress, b) Detailed description of DEGs related to ABA pathways which were induced due to alkalinity stress. PUB44: Promotes the degradation of AAO3 and thus represses abscisic acid (ABA) biosynthesis; XERICO: Function on ABA homeostasis at post-translational level, probably through ubiquitin/proteasome-dependent substrate-specific degradation; AHK3: Redundant negative regulator of ABA signalling; ABC25G: High affinity ABA transporter that mediates export of ABA, with a preference for ( +)-ABA, through the plasma membrane, especially in vascular tissues (e.g. phloem companion cells), and is involved in the intercellular ABA signalling pathway; PTR19: Mediates cellular ABA uptake; DHN1: Induced by ABA; LST8: Involved in plant growth; RGLG5: Mediates ubiquitination and subsequent proteasomal degradation of the target protein PP2CA, a major inhibitor of ABA signalling; WRKY24: Negative regulator of ABA signalling via specific repression of ABA-induced promoters; NAC2: Transcription factor that activates the expression of senescence and ABA associated genes including NCED1, ABCG40, CYP707A2, SAG113, SGR1 and PAO, by directly binding to their promoters; JA2: Regulates the expression of NCED1, and required for the stomatal closure. Both the images are created with BioRender.com

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BMC Plant Biology

ISSN: 1471-2229

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